How Does EDTA Chelation Works?

How Does EDTA Chelation Works?

EDTA therapy has multiple mechanisms of action that positively affect arterial plaque formation and cell membrane function. EDTA binds or chelates with the overabundant or offending mineral. Technically, chelation is defined as “the incorporation of a metal ion into a heterocyclic ring structure.” It is this binding action that is so valuable in removing and intended substance from the circulatory system or other parts of the body. The substance is then disposed of mainly via urine. Multiple studies have shown that EDTA is successful in removing calcium from blood, arterial plaque, arterial walls and other body areas. (As for as removing needed calcium from bones and related areas, there are studies that show EDTA does not bind with significant amounts.)

Other studies have shown EDTA’s ability to effectively fight free radicals scavengers, which can be very important in maintaining coronary and vascular health. There is data pointing to and increase in free radical activity after primary coronary angioplasty in acute myocardial infraction. Because chelating agents can offer control free radical activity, they appear to be useful agents to improve the outcome in patients with coronary artery disease. Also, it appears that free radicals cause the linings of the blood vessel walls to become irritated, causing the body to send calcium to aid in the healing of the irritated area. As a result, the calcium binds with cholesterol, fats, scar tissue and other debris to build a debilitating coating over the affected area. It is this buildup that is referred to as arterial plaque.